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10.1038/s41568-018-0050-3 4 CastoldiF.HyvnenM

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A landmark single-cell study of human dilated cardiomyopathy integrating tens of thousands of cells/nuclei showed a striking pattern: failing-heart cardiomyocytes converge on a shared stressed program (including fetal-gene re-expression), while non-myocytes diversify into HF-associated subpopulations absent in normal hearts ( Multi-omic profiling is also uncovering novel drivers and repurposing opportunities

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P53 Activation as Novel Therapeutic Strategy for AML