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A review of aerobic exercise effects on cognitive function and neuro-physiological underpinning mechanisms

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[23] Most long-term effects are caused by damage to DNA

507 The brains of individuals with preclinical AD (PCAD) and AD exhibit oxidative damage to a variety of molecules, e.g., accumulation of protein carbonyls (PCs) in regions that are rich in A-peptide-containing SPs, increased lipid peroxidation in AD and PCAD hippocampi and elevated DNA damage

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This convergence renders the NF-B and C/EBP pathways promising drug targets for the modulation of the SASP