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Mitochondrial and mitochondrial-independent pathways of myocardial cell death during ischaemia and reperfusion injury

PB: Supervision, Writing original draft, Writing review & editing

Excessive ROS levels can induce cellular damage, initiating a cascade of effects that potentially culminate in tissue and organ injury ( 3.1.1 Impact of ROS on cellular function and atherosclerosis Elevated ROS can induce endothelial cell dysfunction or death, disrupting the vascular barrier and leading to vasoconstrictive and vasodilatory dysfunction, vascular smooth muscle cell proliferation and migration, inflammatory responses, and thrombosisprocesses intimately associated with various cardiovascular disorders, including atherosclerosis, hypertension, and CHD ( in vivo ROS generation represents a promising therapeutic strategy for CHD treatment

Central to this is the role of probiotics in balancing gut bacteria, which in turn affects digestion, immunity, and nutrient absorption

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doi:10.1053/j.gastro.2019.09.041 85Park SC, Jeen YT