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Reducing PLOOH buildup shields cells from ferroptosis, delaying the quick and irreversible destruction to membrane components [36, 37]

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Notably, epithelial-mesenchymal transition (EMT) activity was enriched in both the M1 cluster and post-treatment metastases, while MYC activity was more prominent in pretreatment metastases and the M2 cluster, suggesting that these metastatic phenotypes may be influenced by treatment exposure

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When comparing patients with low and normal TBS, no significant differences were found: RF55.9 [11.7

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