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Inhibition of GPx1 activity partially normalized the cardiomyopathy in Hsp27 transgenic mice, suggesting that reductive stress induced by excess GSH levels and GPx1 activity contributes to cardiac dysfunction in this model (119)

Although current results show some heterogeneity, hypoxic preconditioning remains a potential strategy to improve stem cell transplantation adaptability

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Previously, upregulation of GST genes was observed in treated A

Such differential targeting of subcellular organelles subsequently resulted in a cell functional change