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As melanin synthesis generates ROS, aging leads to a decline in antioxidants like catalase, glutathione, and SOD, creating an imbalance

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Biochemically, once ferroptosis is activated, it cannot be suppressed by apoptosis, necrosis, or autophagy inhibitors but can be hidden by iron chelators such as deferoxamine (DFO) or antioxidants such as Trolox and fer-1

This pattern could reflect either a disruption to GSSG-mediated redox homeostasis or compensatory enhancement of the antioxidant defense system under OS conditions

[DOI] [PMC free article] [PubMed] [Google Scholar] Yamamoto S., Watanabe B., Hiratake J., Tanaka R., Ohkita M., Matsumura Y

I105V polymorphism and promoter methylation of the GSTP1 gene in prostate adenocarcinoma