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Therapeutic targeting of proteostasis in metabolic diseases Targeting HSPs in metabolic diseases Given their central roles in metabolic homeostasis, heat shock proteins (HSPs) have emerged as attractive therapeutic targets in metabolic disease

One trial involving six courses of Epithalamin over three years demonstrated decelerated cardiovascular aging, preserved physical endurance, normalized circadian melatonin rhythm, and improved carbohydrate and lipid metabolism

Excessive cyanide can be highly toxic by binding to the ferric ion (Fe 3+ ) of complex IV, thereby inhibiting oxidative metabolism and ultimately causing cell death [20,21]

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and decreased levels of proinflammatory cytokines

Situations with an excess uptake of lipids by the liver require an adaptive increase in mitochondrial oxidative capacity