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Iron chelation regulates cyclin D1 expression via the proteasome: a link to iron deficiency-mediated growth suppression

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Phase II trial of acivicin versus etoposide-cisplatin in non-small cell lung cancer: an Eastern Cooperative Oncology Group study

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In addition to the classical RSL3, there is also ML162, a drug with a similar mechanism of action to RSL3, and C18, as a derivative optimized based on RSL3, not only has a significantly higher inhibitory activity, but also has a better in vivo antitumor effect and lower toxicity, C18 may be a promising covalent inhibitor of GPx4 that induces ferroptosis for the treatment of triple-negative breast cancer