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The investigators reported that LR3-IGF-1 produced greater anabolic effects than native IGF-1 across both administration modes, with the differential most pronounced under once-daily injection conditions

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or oxidizing agent: ChT)

Consequently, the further inhibition of Gln metabolism and fatty acid metabolism through the suppression of ASCT2 expression, which poses a significant threat to the energy supply required for cancer cell proliferation, emerges as a promising anticancer target