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The absence of smad4 was also found to enhance mitochondrial ATP synthase inhibitory factor (ATPIF1) activity, ATP synthase inhibition, and reduce mitochondrial membrane potential, ultimately promoting glycolysis by decreasing glucose metabolic flux and increasing glucose accumulation in the podocyte [62]

We conclude that the future of ADCs in CRC lies in their rational application as immune-priming agents, creating powerful synergies in combination with checkpoint inhibitors to break therapeutic resistance and durably improve patient outcomes

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FIGURE 2 gsnor Plants Have Higher GSH-Dependent Antioxidant Capacity Resistance against oxidative stress is often related to an enhanced cellular antioxidant capacity

Cheng et al., 1993

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