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Findings that would support a primary metabolic defect in copper metabolism include a previously recognized breed predisposition, copper accumulation that precedes cholestasis or inflammation, centrilobular (zone 3) distribution of copper, histochemical score for copper of 3+ or greater, or quantitative copper measurements that exceed 2000 g/g dry weight.54, 57, 66 Special stains of the liver should be requested to evaluate for infectious agents such as leptospirosis

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pmid:22474993 53

Check whether TB-500 is actually being studied or whether the paper used full-length thymosin beta-4

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