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Noticeably, tiny iron oxide structures enhance Beclin-1 production, leading to increased ATG5 levels, promoting autophagy-triggered ferroptosis in the treatment of glioblastoma ( Table 3 ) (310)

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4,5 A key advantage in the study of APAP toxicity is that its human pathophysiology can be effectively reproduced in a mouse model in vivo and in primary mouse hepatocytes

Pulmonary function test of mice Intraperitoneally anesthetized mice with tribromoethanol (0.2 mL/10 g) and subsequently fixed in the supine position on a constant temperature platform maintained at 37 C

It is of interest that the strongest deterioration of the esophageal mucosa exposed to acid + pepsin solution was observed following inactivation of sensory nerves with capsaicin

Hypoxia-induced signaling in the cardiovascular system: Pathogenesis and therapeutic targets