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Mechanistic studies indicate that ATR-I exerts anti-inflammatory effects by regulating NLRP3 inflammasome-associated signaling pathways: it significantly reduces levels of key pro-inflammatory factors NO and TNF-, while suppressing expression of inflammatory mediators such as MCP-1 and MMP-9 in microglia, thereby exerting its anti-inflammatory neuroprotective effects ( Tripterygium wilfordii extract (10 g/kg, administered for 7 days) effectively prevents the loss of dopaminergic neurons in the substantia nigra and maintains the function of the nigrostriatal pathway, while significantly improving motor deficits in MPTP-induced PD mouse models

It is studied in experimental models of skin wounds, tendon and ligament injuries, muscle degeneration, gastrointestinal inflammation, lung damage, and neuroinflammatory conditions

IDH1 mutations are common genetic abnormalities in human malignancies

One of its most profound benefits is its ability to combat neuroinflammation and alleviate the debilitating brain fog associated with Long COVID and ME/CFS

R., McFall-Ngai, M

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