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proposed that the sulforaphane-mediated inhibition of differentiation into plasma B and germinal centre B cells may be the mechanism underlying its anti-RA action

[92] demonstrated that organelle-targeting CQDs could preferentially accumulate within mitochondria, the Golgi apparatus, and the endoplasmic reticulum, illustrating how variations in precursor composition and surface functional groups can influence intracellular localisation and enhance organelle-specific imaging contrast [92]