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Neurology 91, e463e471

To rule out that the less protein-membrane interaction observed in hyh mice resulted from any physical or biochemical PM alteration associated with hyh phenotype, we performed a crossover protein-lipid interaction experiment

Rapid determination of reduced and oxidized glutathione levels using a new thiol-masking reagent and the enzymatic recycling method: application to the rat liver and bile samples

13 Becerril-OrtegaJ.BordjiK.FrretT.RushT.BuissonA

Figure 1 Iron metabolism At present, the exact molecular mechanism of iron overload leading to heart damage is mainly considered to be ferroptosis ( 2+ uniporter, which is essential in causing mitochondrial dysfunction and ferroptosis in cardiac iron overload ( Hepcidin, a peptide hormone originated from the liver, regulates iron absorption and distribution by suppressing the activity of transferrin (59)

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