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For example, an enlargement in the labile iron pool in colonic macrophages in the DSS-model of colitis could translate to priming and susceptibility for ferroptotic cell death [38]

The control group was always composed of cells treated with an equal volume of control M-CFS (without the mushrooms)

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In 1988, Correa initially proposed a disease model elucidating the malignant transformation from H

Mitochondrial autophagy-related gene signatures associated with myasthenia gravis diagnosis and immunity