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Here, we identified a critical role of mitochondria in mediating oxidative stress-induced ferroptosis in cardiomyocytes

And that I think what explains that is just the vicious cycle of the mitochondria got, they lost a quarter percent here or a quarter percent there, and they just started repairing themselves less effectively because theyre the engines fueling everything, including that

The appreciation we receive from patients and their families, knowing that we have improved and preserved their quality of life, is an unparalleled reward

doi: 10.1161/HYPERTENSIONAHA.109.144964 122 CrowleySDZhangJHerreraMGriffithsRRuizPCoffmanTM

There's no room for improvisation here

Safety in controlled studies: Minimal systemic hormonal effects simplify interpretation of fat-specific pathways