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Conclusion and perspectives Copper functions as a double-edged sword within cells: it is an essential cofactor for numerous enzymes that facilitate tumor progression, yet its excess can lead to cuproptosis, resulting in cellular demise

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The relative contribution of this pathway to disulfide formation is not clear but it is known that combined knockdown of Ero1, PrxIV, and VKOR compromises disulfide formation and causes cell death in a mammalian cell culture system (Rutkevich and Williams 2012)

Microbiol 12 , 721691 (2022)

This paradigm shift in peptide engineering combines data-driven insights with traditional rational design principles, substantially expanding the landscape of feasible peptide therapeutics

Targeting the prostate for destruction through a vascular address