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demonstrated that improving mitochondrial metabolism, reducing oxidative stress and mitochondrial fission via the deletion of DNA-dependent protein kinase catalytic subunit (DNA-PKcs), thus alleviating sepsis-mediated myocardial injury, liver injury, and kidney injury in a sepsis model

For both metabolomics and tracing experiments, treated CD8 + T cells were collected and washed with cold saline solution twice in 15 mL tubes

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BCAT1E61A's higher enzymatic activity can promote the catabolism of BCAA and accelerate cell growth and metastasis, and BCAT1 can also activate the PI3K/AKT/mTOR pathway to contribute to the development of tumors [40]