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however, in KEAP1-mutated lung cancer cells, the upregulation of NRF2 results in increased FSP1 expression, thereby facilitating ferroptosis induction [101]

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This is for several reasons, enumerated in detail in a recent review [1], partly relating to problems with drug uptake into the worm

Therapeutic strategies that modulate apoptosis may interfere with or promote regulated necrosis

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Since FAO is increased in a hypoglycemic state, angiogenesis may also be increased due to compensatory VEGF expression