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The accumulation of protein aggregates and damaged organelles due to defective autophagy contributes to neuronal dysfunction and degeneration

The nucleophilic GSH attacks the electrophilic substrate forming a thioether bond between the cysteine residue of GSH and the electrophile

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S100B is increased in Parkinsons disease and ablation protects against MPTP-induced toxicity through the RAGE and TNF-alpha pathway