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Aging both impedes the uncontrolled growth of damaged cells and supports their removal by paracrine signals

Under conditions of iron depletion or chelation, PHD activity is impaired, resulting in HIF-1 stabilization even in normoxia [89]

Furthermore, when using RIPK3 as a therapeutic target for NASH, the patient population must be carefully selected, as its inhibition may exacerbate insulin resistance and intolerance in obese individuals [141]