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Vitamin D, essential minerals, and toxic elements: exploring interactions between nutrients and toxicants in clinical medicine

This method of utilizing disease-specific interactions presents an appreciable advancement in specificity and relevance (66)

Specifically, lipid peroxidation linked to ferroptosis and mitochondrial dysfunction associated with cuproptosis both have the potential to amplify inflammatory cascades

In sepsis-associated encephalopathy, mitochondrial dysfunction occurs in the setting of systemic inflammation, which contributes to increased blood-brain barrier permeability and resultant neuroinflammation [15, 16]

The absorbed glutamine is subsequently catabolized into the TCA cycle, providing essential energy for cellular functions [29] (Fig