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Description

Chen LYC, Biggs CM, Jamal S, Stukas S, Wellington CL, Sekhon MS

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When IGF-1 (or an agonist analog such as IGF-1 LR3) binds the alpha subunits, the receptor undergoes a conformational change that activates the intrinsic tyrosine kinase domains of the beta subunits, triggering autophosphorylation and the recruitment of docking proteins