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( 10.1001/jama.2019.18714) [DOI] [PMC free article] [PubMed] [Google Scholar] Schnemann H, Broek J, Guyatt G, Oxman A.2013

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On the other hand, oxidative stress leads to an increase in the release of cytochrome C from the mitochondria which activates the apoptotic mechanism in the cells by activating caspases (caspase-3/7, 8 and 9) and pushing the cells to the programmed cell death

Generation of superoxide anion by the NADH dehydrogenase of bovine heart mitochondria

Funding This research was funded by the Research Vice-Chancellor of Tabriz University of Medical Sciences (Grant number: 59360)

Electrophysiological and phenotypic profiles of taxane-induced neuropathy