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376 The causal role of oxidative damage in the ageing process remains controversial, partly because of the absence of a clear correlation between the efficacy of antioxidant defenses and extended cellular function or longevity

This phenomenon supports the notion that increased oxidative stress is an important pathogenic mechanism of HCC [2,3,5], and suggests that GSH might be utilized and oxidized to GSSG in order to cope with increased oxidative stress during the development of HCC

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In probing the mechanisms of ferroptosis within the hepatic microenvironment, we question whether such mitochondrial anomalies are a driving force or simply a corollary of ferroptosis