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Glutathione reductase activity maintains the GSH concentration in cells and thus is expectedly high for Lc

28,52,53 Over the past 5 years, a growing body of works have extended this concept to non-malignant inflammatory diseases (eg

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Common structural features of MAPEG a widespread superfamily of membrane associated proteins with highly divergent functions in eicosanoid and glutathione metabolism

Human Fetal TNF-Alpha-Cytokine-Producing CD4(+) Effector Memory T Cells Promote Intestinal Development and Mediate Inflammation Early in Life

3d), suggesting that they are either exhibiting unique functions on unknown substrates or regulating FSP1 transcription indirectly