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Arutjunyan, A., et al

Owing to the lack of robust clinical data, clinicians and healthcare systems may not be equipped to identify or manage adverse effects associated with these molecules

Micke, P., Beeh, K

Circulation 91 , 359364 (1995)

Rheumatology 62 , 12641271 (2023)

To attenuate the disruption to energy homeostasis in cells undergoing excessive autophagic flux, activation of the transcription factors TFEB and TFE3 promotes the expression of FAM134 family proteins and TEX264, and the regulation of casein kinase 2 (CK2) mediated via phosphorylation enhances the affinity of autophagy receptors for LC3 (Fig