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In gemcitabine-resistant cells, miR-200 and miR-21 exhibit elevated and diminished levels, respectively

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This work was supported by institutional grants from the Institutional Animal Ethics Committee (IAEC) with registration no

Mechanistically, BPC-157 appears to function through nitric oxide-mediated vasodilation, angiogenic factor regulation, and recruitment of fibroblasts and endothelial progenitor cells 1-3

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