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Mechanisms involved in AMPK-mediated deposition of tight junction components to the plasma membrane

Why 200-350, and not 0.1, 5, or 5,000

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This activation initiates a signaling cascade that turns on Protein Kinase C (PKC) and Mitogen-Activated Protein Kinases (MAPKs), which dynamically stabilize the actomyosin ring, pulling the tight junction proteins firmly back into place and sealing the paracellular gaps

GSTM2 was highly up-regulated in hepatic steatosis tissues and high-fat diet (HFD) fed mice

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