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Immunologically, ferroptosis leads to the release of damage-associated molecular patterns, initiating inflammatory responses [110]

It may be that increased cysteine impedes the synthesis of its precursor, serine, to control SAA levels

However, this can be challenging due to variations in size, structure, and overall appearance which are further impacted by changes linked to disease severity

Emerging evidence has demonstrated that DA neurons come in various types that send distinct motivational messages about rewarding and non-rewarding events [42]