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Manganese upregulates cellular prion protein and contributes to altered stabilization and proteolysis: relevance to role of metals in pathogenesis of prion disease

instead, it gains a new ability to facilitate the NADPH-dependent reduction of -KG to D-2-hydroxyglutaric acid (D-2HG), an oncometabolite

Microglial NF-B drives tau spreading and toxicity in a mouse model of tauopathy

Integration of anatomical and radiological analysis suggests more segments in the human kidney

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Receptor activation occurs wherever IGF-1 receptors are expressed