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However, a study by A

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Side effects of ferroptosis-based antitumor therapy There is growing evidence that current ferroptosis activators can trigger cell death in normal cells, resulting in adverse effects during cancer therapy

Their relatively low molecular weight, while beneficial for tissue penetration, also facilitates rapid renal clearance, curtailing plasma persistence

The risk is not zero, but the margin is wider

In resting cells, low levels of NIX and BNIP3 are maintained by ubiquitination and proteasomal degradation through an OMM-localized protein called SCFFBXL4 E3 ubiquitin ligase complex (85)