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This identified that TIGAR-mediated lysosome repair may be a potential intervention for neurodegenerative diseases such as PD

Histone deacetylase inhibition via RGFP966 releases the Brakes on sensory cortical plasticity and the specificity of memory formation

[DOI] [PMC free article] [PubMed] [Google Scholar] 48.Moura C.S., Lollo P.C.B., Morato P.N., Risso E.M., Amaya-Farfan J

Fact: Antioxidants work best in a network

Therefore, GH maintains triglyceride storage in the liver through three mechanisms, inhibiting intrahepatic triglyceride lipolysis, inhibiting lipid oxidation, and enhancing lipogenesis (Vijayakumar et al., 2011)

Therefore, there is an urgent need for alternative therapeutic approaches, including plant-derived phytochemicals, that can directly modulate the core pathogenic mechanisms of PD, such as neuroinflammation, mitochondrial dysfunction and S aggregation