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Alterations in energy metabolism in cardiovascular diseases often involve systemic pathophysiological changes across multiple systems, while the key energy metabolic targets (AMPK, SIRT) exhibit functional variances in different tissues, suggesting the need for in-depth mechanistic exploration using tissue-specific animal models and expanding clinical research in the future

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Furthermore, our findings suggest that continuous monitoring of pgRNA levels after treatment may hold predictive value for the recurrence of advanced HCC

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