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Thus, copper bound to the mitogen-activated protein kinase kinase 1 (MEK1) enhances its phosphorylation, thereby promoting tumorigenesis [4]

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As expected from our previous experiments, the P44A mutant precipitated slower than the WT in the presence of EGCG, although the effect was not as pronounced as in the solid-state NMR experiments reported above

doi: 10.1016/j.immuni.2022.05.014

These findings indicate that inflammatory conditions associated with the overactivation of NF-B may misalign the molecular clock, thereby altering the transcription of several target genes and contributing to metabolic inflammation through direct transcriptional reprogramming