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We suggest that reduced GPx1 in monocytes recruited to the gut and intestinal macrophages renders these cells vulnerable to reactive oxygen species-evoked ferroptosis cell death and that unraveling the participation of this pathway in Crohns disease may reveal novel therapeutic approaches to this chronic condition

Neuroinflammation 19:262

however, as mentioned above, GM-CSF indeed exerts a pathological function in the phase of CS, implying that blocking GM-CSF signaling may achieve clinical benefits in COVID-CS

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and better control of medical costs (La Thangue and Kerr, 2011)

Kositamongkol, U