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doi: 10.3389/fimmu.2026.1758379 Received 01 December 2025 Revised 05 March 2026 Accepted 05 March 2026 Published 28 April 2026 Volume 17 - 2026 Edited by Varun Sasidharan Nair, Italian Institute of Technology (IIT), Italy Reviewed by Mahmoud Abdelrahman Alkabbani, Egyptian Russian University, Egypt Lingjie Meng, Zunyi Medical University, China Updates Copyright 2026 Sivalingam

Huang, J, Tan, KS, Altorki, N, Antonoff, MB , Blackmon, SH, Bueno, R, Burt, BM, Demmy, TL, Evans, N, Donahoe, L, Harpole, DH, Jarrar, D, Kozower, BD, Lanuti, M, Liberman, M, Lin, J, Liou, D, Liptay, MJ, Luketich, J, Pennathur, A, Petersen, G, Ripley, RT, Rochefort, M, Seder, CW, Shrager, JB, Su, S, Tong, BC, Shargall, Y, Vaporciyan, AA, Waddell, TK, Weksler, B, Wigle, DA, Yendamuri, S, Jones, DR

Although NAC is often used in the clinic to restore hepatocellular GSH levels to protect against APAP hepatotoxicity, in the context of mitochondrial cholesterol accumulation, which impairs the transport of cytosol GSH into mitochondria, the use of NAC may be inefficient to specifically replenish mGSH stores, requiring the use of permeable GSH prodrugs such as GSH ethyl ester (GSHEE)

A kinetic analysis of the reactions of hypobromous acid with protein components: implications for cellular damage and the use of 3-bromotyrosine as a marker of oxidative stress