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Dual Inhibition of CDK4/6 and XPO1 induces senescence with acquired vulnerability to CRBN-based PROTAC drugs

Neuroprotection by dimethyloxalylglycine following permanent and transient focal cerebral ischemia in rats

Studies have also demonstrated that mice expressing ApoE4 in tauopathy models exhibit more severe tau-induced neurodegeneration compared to other isoforms, while mice lacking ApoE are protected from this effect (Shi et al., 2017)

10:e2960

The dysregulation of glycolysis in these cells has been identified as a significant initiator of neuronal impairments in AD [66]

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