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The by-products of free radical damage to polyunsaturated fatty acids react with thiobarbituric acid to form products that may be assayed as an index of oxidative damage and lipid peroxidation (Armstrong & Browne, 1994)

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Defects in intestinal mucosal antioxidant defense constitute the initiating factor in the pathogenesis of IBD, with oxidative imbalance in the intestinal mucosa further associated with disease activity and progression

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