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In contrast, LCN2 deletion improves fat and muscle mass mainly through appetite regulation, whereas INSL3 elevation induces anorexia that precedes adipose tissue wasting that, in turn, anticipates muscle atrophy (Olson, et al., 2021a

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This elimination of the gbu gene cluster from the gut microbiome may be a mechanism that helps explain why plant-based diets are associated with reduced risk of CVD and other metabolic disorders associated with elevated TMAO.14 Researchers have questioned whether GBB levels themselves could play a role in CVD risk