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elegans (having mutations in NADH-ubiquinone oxidoreductase) where riboflavin supplementation enhanced the assembly of complex I and IV that further resulted in reduced oxidative stress and increased metabolic functions [39]

The low response rate of lenvatinib, which is mostly caused by resistance development, limits its practical utility in treating patients with advanced liver cancer, even though it is the recommended initial treatment for this condition

To this end, we computed additive genetic associations of the two ratios with 5.55 mio autosomal SNPs at each time point, while adjusting for age, sex, time point-specific HRSD 17 score, the first five PCs to account for population stratification

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Ikubo Y, Sanada TJ, Hosomi K, Park J, Naito A, Shoji H, et al