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However, excessive ROS production following ablation of de novo glutathione (GSH) synthesis suppresses the activity of mammalian target of rapamycin and the expression of transcription factors NFAT and c-MYC, the latter of which control metabolic reprogramming following T cell activation (15, 47, 48)

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10.3390/molecules27082439 203 YamamotoM.KenslerT