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The covalently bound drug is then transported to the tumor tissue, where it accumulates via two mechanisms: 1) cancer cells have been demonstrated to use albumin as an amino acid source and, thus, show an increased uptake of this plasma protein 15,16 , and 2) the enhanced permeability and retention (EPR) effect, which enables macromolecules to enrich in cancerous tissue, due to disordered and thus fenestrated vascularization and impaired lymphatic drainage 17

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These coenzymes perform distinctly different roles in metabolism, despite their similar structures

Several studies have reported the role of ferroptosis in microglia, which put forward by Wu et al., 2020)