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For example, miR-139 increases the radiosensitivity of NSCLC by directly targeting cJUN and KPNA2, disrupting the NRF2 pathway and amplifying radiation-induced lipid peroxidation (122)

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As such, CRY1 emerges as a pro-tumorigenic factor that rhythmically controls DNA repair mechanisms and cell survival through temporal transcriptional regulation 27

These disruptions can impair synaptic function by interfering with proper intracellular trafficking, signaling, and communication in neurons, contributing to the early pathology of sporadic AD (Marquer et al., 2014

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