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Loss of normal SQOR function directly disrupts hydrogen sulfide (H 2 S) homeostasis, leading to mitochondrial dysfunction, and also exacerbates redox imbalance in the kidney, triggers innate immune activation, and reshapes cellular metabolism (for example, by affecting protein stability and causing excessive ubiquitin-mediated degradation)

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Understanding variants in genes affecting fibroblast function and inflammatory response may support a more informed conversation with your provider about whether GHK-Cu or other peptide-based approaches align with your individual biology and wellness objectives