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doi:10.3389/fendo.2020.00641 Pasquali R

Starting at a low dose and increasing gradually helps minimize initial side effects

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Participants A total of 46 patients with active RA, fulfilling the 2010 RA classification established by "American College of Rheumatology /European League Against Rheumatism" (Aletaha et al

Firstly, as will be discussed below, LCACs induce Ca 2+ overload within cardiomyocytes via release of sarcoplasmic reticulum (SR) Ca 2+ and inhibition of mitochondrial Ca 2+ uptake ( 2+ overload effect has been shown to depend on LCAC fatty acyl chain length, suggesting that longer LCACs have greater membrane penetration and, therefore, cause greater membrane disruption ( Opposing these findings of membrane perturbation and potentially irreversible sarcolemma disruption are the reports of reversible electrophysiological and inotropic effects following exogenous LCACs wash out or after/during normoxic reperfusion post ischemia ( In sum, the evidence strongly supports membrane perturbation and altered phospholipid packing in mediating the electrophysiological and inotropic effects of LCACs in the heart