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GIP has been shown to decrease food intake while blunting the metabolic adaptive responses that usually occur with calorie restriction resulting in weight reductions, and when combined with GLP-1 receptor agonism, may result in greater effects on markers of metabolic dysregulation such as body weight, glucose and lipids

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In addition, cerebral inflammation is known to be a key component of some neurologic diseases including Alzheimers disease, multiple sclerosis, and migraine (19)

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Some of these physiological adaptations can favor weight regain by increasing hunger and promoting energy storage

Specifically, we highlight the latest research on the HGF/c-Met axis, including the roles of non-coding RNAs in regulating of liver regeneration by HGF