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Alpha-1 antitrypsin deficiency affects approximately 1 in 2,0006,000 individuals globally, with an estimated 3.4 million people harboring severe deficiency alleles and over 116 million carriers worldwide [20, 21]

The enhanced autophagy in drug-resistant cells is mainly due to the destruction of mitochondrial function caused by tamoxifen, and drug-resistant cells meet their energy demand through autophagy

Currently, activating key innate immune pathwaysparticularly the cyclic GMP-AMP synthase-stimulator of interferon genes (cGAS-STING) pathwayhas emerged as a promising strategy to enhance antitumor immune responses [8,9,10,11,12,13,14,15,16,17]

IR and an excessive influx of FFAs drive an overload of fatty acid -oxidation, increasing ROS and causing a loss of mitochondrial membrane potential (m)

Ive found that most older patients prefer oral supplements over regular vitamin B12 injections, which is understandable