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This abnormal mechanism promotes malignant transformation through dual effects: on the one hand, it simultaneously inhibits key tumor suppressor genes, and on the other hand, it activates oncogenic signaling pathways, ultimately leading to genomic instability

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Pulmonary rehabilitation following exacerbations of chronic obstructive pulmonary disease

TGF-1 is well-known not only to trigger mitochondrial ROS formation but also to activate genes related to EMT (Ishikawa et al

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